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    indomethacin
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    Melatonin And Heart Disease

    In 1995, a study which appeared in Lancet (Brugger, et al.) looked at nocturnal melatonin levels of 15 patients with documented coronary heart disease vs. 10 controls without heart disease. The researchers found that daytime melatonin levels were low in both groups. However, at night the group of patients with coronary heart disease had five times lower the concentration of melatonin than the healthy control group.

    The researchers concluded that further research should be carried out to see if supplemental melatonin can prevent development of atherosclerosis, and coronary heart disease.

    It is also interesting to observe that most of the fatal heart attacks occur in the morning, when melatonin levels are low (6:00 A.M.-noon) (Muller, et al., 1985). Leach, et al., (1980), have demonstrated that platelet aggregation and thromboxane release is inhibited by melatonin and that this action is similar to indomethacin through inhibition of prostaglandin 2 synthesis (PG2).

    Platelet aggregation is a well recognized mechanism of coronary thrombosis. During the night when melatonin levels are high the blood pressure and pulse are low, which is beneficial in prevention of coronary heart disease.

    In addition, melatonin was shown to lower cholesterol levels which is another risk factor in developing atherosclerotic heart disease and strokes (Mori,et al., 1989; Mori, et al., 1984; and Aoyama H, et al.,1988).

    Muller-Wieland D., et al., (1994), have demonstrated that melatonin blocked cholesterol production by 38% and reduced LDL accumulation by 42%.

    My personal experience and the experience of other researchers using melatonin show that melatonin administration lowers serum cholesterol and in particular LDL-cholesterol. This decreases the risk of atherosclerotic heart disease and stroke.


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